I like the comparison, and while I wrote my comment I have to admit I was
also thinking of bacteria and virusses as an exception: There the speed of
replication can be an important factor for survival. Less DNA means faster
replication and therefore the pressure is on removal of junk DNA. It can
be disputed however that removal of 'junk sourcecode' is a survival factor
for the linux kernel but the benefit may be disputable as well.
Maybe we can elaborate a bit on this comparison, just for fun:
I think not the linux kernel alone, but rather the entire Linux OS could
be compared with a cell. The kernel source encodes vital software parts
including the interactions with hardware - the environment for the
computer. Gcc can be compared with the (transcription and) translation
machinery. DNA can be seen as a language that encodes proteins, with
biological functions: Some are vital, including ones that allow
interactions with the environment: The cellular environment is beyond
the membrane. Interactions occur through membrane receptors, channels,
etc.
Interaction between proteins can be compared with functions selectively
calling other functions. Activation of certain proteins can cause a
cascade of protein interactions, comparable with function calls in a
loop: the activated protein activates particular protein(s) several
times. Some proteins influence intracellular messengers: cellular
global variables.
Transmembrane receptors responding to extracellular signals transmit
this through conformational changes across the membrane to the
intracellular region: These structural changes may allow interactions
with new proteins. Maybe comparable with a combination of hardware
interrupts, signals and the userspace?
The response to extracellular signals often depends on several
sequential interactions between proteins. This provides a protective
layer that can be compared with the kernelspace layer. This is where
the comparison probably becomes too biased to continue.
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